Please use this identifier to cite or link to this item: http://hdl.handle.net/2445/18656
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dc.contributor.authorSáiz Calahorra, Juan Carloscat
dc.contributor.authorSans i Cuffí, Miquelcat
dc.contributor.authorMas Comas, Ana M.cat
dc.contributor.authorOlmedo Casas, Evacat
dc.contributor.authorForns, Xaviercat
dc.contributor.authorLópez Labrador, Francesc Xaviercat
dc.contributor.authorRestrepo Gutiérrez, Juan Carloscat
dc.contributor.authorCosta i Camps, Josepcat
dc.contributor.authorSalmerón Bargo, Juan Manuelcat
dc.contributor.authorGuilera Sardà, Magdacat
dc.contributor.authorAmpurdanés, Sergicat
dc.contributor.authorSánchez Tapias, José M. (José María)cat
dc.contributor.authorJiménez de Anta Losada, María Teresacat
dc.contributor.authorRodés, J.cat
dc.date.accessioned2011-07-07T12:30:30Z-
dc.date.available2011-07-07T12:30:30Z-
dc.date.issued1997-
dc.identifier.issn0017-5749-
dc.identifier.urihttp://hdl.handle.net/2445/18656-
dc.description.abstractBackground¿RNA sequences of the recently identified hepatitis GB virus C (HGBV-C), also named hepatitis G virus (HGV), have been detected in patients with idiopathic fulminant hepatic failure (FHF) but the role of this agent in the disease remains controversial. Aims¿To investigate the presence and implications of HGV infection in a large series of Spanish patients with FHF. Patients¿Sixty eight patients with FHF, including 19 with idiopathic disease, were studied. In 28 cases, studies were performed before and after liver transplantation. For comparison 200 volunteer blood donors and 22 patients transplanted for chronic liver disease were also studied. Methods¿HGV RNA was measured in serum by reverse transcriptase polymerase chain reaction of the 5' non-coding region. Results¿Evidence of HGV infection was found in 3% (6/200) of blood donors and in 19% (13/68) of patients with FHF. HGV infection was more frequent in patients with hepatitis B (24%, 6/25) or hepatitis D (42%, 5/12), than in patients with idiopathic disease (11%, 2/19). Half of the patients with HGV infection used illicit intravenous drugs. Specific clinical features associated with HGV infection were not identified. A very high rate of infection with HGV was observed in patients who underwent liver transplantation, either for FHF (60%, 15/24) or chronic liver disease (45%, 9/20). Conclusions¿In our geographical area, HGV infection is relatively frequent in FHF, but it does not seem to play a major role in idiopathic cases.eng
dc.format.extent4 p.-
dc.format.mimetypeapplication/pdf-
dc.language.isoengeng
dc.publisherBMJ Groupeng
dc.relation.isformatofReproducció digital del document publicat a: http://dx.doi.org/10.1136/gut.41.5.696cat
dc.relation.ispartofGut, 1997, vol. 41, núm. 5, p. 696-699-
dc.relation.urihttp://dx.doi.org/10.1136/gut.41.5.696-
dc.rights(c) BMJ Publishing Group Ltd and British Society of Gastroenterology, 1997-
dc.subject.classificationVirus GB Ccat
dc.subject.classificationHepatitis Gcat
dc.subject.otherGB virus Ceng
dc.subject.otherHepatitis Geng
dc.titleHepatitis G virus infection in fulminant hepatic failureeng
dc.typeinfo:eu-repo/semantics/article-
dc.typeinfo:eu-repo/semantics/publishedVersion-
dc.identifier.idgrec161960-
dc.rights.accessRightsinfo:eu-repo/semantics/openAccess-
Appears in Collections:Articles publicats en revistes (Medicina)
Articles publicats en revistes (Fonaments Clínics)

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