Please use this identifier to cite or link to this item: http://hdl.handle.net/2445/69464
Title: Insulin resistance compensation: not just a beta cell matter?
Author: Montanya Mias, Eduard
Keywords: Resistència a la insulina
Illots de Langerhans
Insulin resistance
Islands of Langerhans
Issue Date: 15-Mar-2014
Publisher: American Diabetes Association
Abstract: The global epidemic of type 2 diabetes is largely secondary to insulin resistance induced by obesity and sedentary lifestyles. Most insulin-resistant subjects are able to increase b-cell secretion to meet the increased insulin demand and do not develop diabetes. However, when b-cell compensation fails, type 2 diabetes develops (1,2). Understanding the mechanisms of this compensatory response is of fundamental importance to elucidate the pathophysiology of type 2 diabetes and has implications for the treatment of the disease.
Note: Reproducció del document publicat a: http://dx.doi.org/10.2337/db13-1843
It is part of: Diabetes, 2014, vol. 63, num. 3, p. 832-834
Related resource: http://dx.doi.org/10.2337/db13-1843
URI: http://hdl.handle.net/2445/69464
ISSN: 0012-1797
Appears in Collections:Articles publicats en revistes (Ciències Clíniques)

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