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cc-by-nc-nd (c) Navarro Brugal, Gemma et al., 2024
Si us plau utilitzeu sempre aquest identificador per citar o enllaçar aquest document: https://hdl.handle.net/2445/218573

Homodimerization of CB2 cannabinoid receptor triggered by a bivalent ligand enhances cellular signaling

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G protein-coupled receptors (GPCRs) exist within a landscape of interconvertible conformational states and in dynamic equilibrium between monomers and higher-order oligomers, both influenced by ligand binding. Here, we show that a homobivalent ligand formed by equal chromenopyrazole moieties as pharmacophores, connected by 14 methylene units, can modulate the dynamics of the cannabinoid CB2 receptor (CB2R) homodimerization by simultaneously binding both protomers of the CB2R-CB2R homodimer. Computational and pharmacological experiments showed that one of the ligand pharmacophores binds to the orthosteric site of one protomer, and the other pharmacophore to a membrane-oriented pocket between transmembranes 1 and 7 of the partner protomer. This results in unique pharmacological properties, including increased potency in Gi-mediated signaling and enhanced recruitment of β-arrestin. Thus, by modulating dimerization dynamics, it may be possible to fine-tune CB2R activity, potentially leading to improved therapeutic outcomes.

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NAVARRO BRUGAL, Gemma, GÓMEZ-AUTET, Marc, MORALES, Paula, REBASSA, Joan-biel, LLINAS DEL TORRENT, Claudia, JAGEROVIC, Nadine, PARDO, Leonardo, FRANCO FERNÁNDEZ, Rafael. Homodimerization of CB2 cannabinoid receptor triggered by a bivalent ligand enhances cellular signaling. _Pharmacological Research_. 2024. Vol. 208, núm. 1-11. [consulta: 27 de novembre de 2025]. ISSN: 1043-6618. [Disponible a: https://hdl.handle.net/2445/218573]

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