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cc-by-nc-nd (c) American Diabetes Association, 2014
Please use this identifier to cite or link to this item: https://hdl.handle.net/2445/69464

Insulin resistance compensation: not just a beta cell matter?

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The global epidemic of type 2 diabetes is largely secondary to insulin resistance induced by obesity and sedentary lifestyles. Most insulin-resistant subjects are able to increase b-cell secretion to meet the increased insulin demand and do not develop diabetes. However, when b-cell compensation fails, type 2 diabetes develops (1,2). Understanding the mechanisms of this compensatory response is of fundamental importance to elucidate the pathophysiology of type 2 diabetes and has implications for the treatment of the disease.

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MONTANYA MIAS, Eduard. Insulin resistance compensation: not just a beta cell matter?. Diabetes. 2014. Vol. 63, num. 3, pags. 832-834. ISSN 0012-1797. [consulted: 16 of June of 2026]. Available at: https://hdl.handle.net/2445/69464

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